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- Table of Contents
, No Gclids
Disease Info Card
Absence Of Sensation
Information about Absence Of Sensation: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Absence Of Sensation
Most recent studies have shown that Absence Of Sensation shares some biological mechanisms with ataxia, atrophy, cerebrovascular-accident, malnutrition, muscle-weakness, muscular-atrophy, neoplasms, nervous-system-disorder, nervousness, neuralgia, numbness, pain, paraplegia, paresthesia, peripheral-neuropathy, polyneuropathy, sensation-disorders, spinal-cord-diseases, weakness.
Among the many pathways, these few ones have gauged particular interests from scientists studying Absence Of Sensation, and have been seen in publications frequently: Aging, Coagulation, Cognition, Dehiscence, Hypersensitivity, Innervation, Localization, Locomotion, Micturition, Muscle Atrophy, Ossification, Pathogenesis, Proprioception, Reflex, Regeneration, Sensory Perception, Sensory Processing, Transport, Transposition, Wound Healing
Quite a number of genes have been found to play important roles in Absence Of Sensation, such as ACAT1, C2, C6, C7, CD1A, COPS2, CSF2, CXCL10, GRIP1, HNRNPC, LAMC2, MMEL1, MPZ, PES1, PLXNB1, PMP22, PSMA7, SLC25A5, SPTLC1, WNK1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.